Back to browse

EXP000833

Paper

N-terminal modification of an LAH4-derived peptide increases mRNA delivery in the presence of serum (2024)

Peptide

LAH4-A1-Sali

Sequence: KKALLAHALHLLAALALHLAHLLKKA

RNA

mRNA

All experiment fields

Experiment Id EXP000833
Paper N-terminal modification of an LAH4-derived peptide increases mRNA delivery in the presence of serum
Peptide LAH4-A1-Sali
Delivery Success Class no
In Vivo Flag no
Uptake Confirmed yes
Label Confidence high
In Vitro Functional Effect yes
Endosomal Escape Evidence yes
Peptide Concentration
Rna Concentration
Mixing Ratio Typically 5 µg peptide per 0.5 µg mRNA (HCT116) or 7 µg peptide per 1 µg mRNA (HEK293T GFP/Cy5 experiments); conditions varied.
Formulation Format peptide/mRNA complexes (polyplex)
Formulation Components Buffers used for complexation included 10 mM acetate pH5, 150 mM NaCl, or 10 mM phosphate buffer pH6.25 (also PBS, HBG in optimization).
Size Nm 257.00
Zeta Mv
Model Scope in_vitro
Model Type in vitro
Cell Lines Or Primary Cells HCT116; HEK293T
Animal Model
Administration Route
Output Type Functional reporter expression (luciferase and/or GFP)
Output Value
Output Units
Output Notes Modified LAH4-A1 peptides evaluated mainly in HCT116/HEK293T (luciferase/GFP); Tyr2 and Sali show improved activity in phosphate pH6.25 and improved serum tolerance. DLS in 10 mM phosphate pH6.25: 257 nm ±219 (PDI 0.218) with 10 µg peptide +0.8 µg FLuc mRNA. Uptake/association shown by flow cytometry with Cy5-labeled mRNA (all cells Cy5+). Endosomal acidification dependence shown: concanamycin A (H+-ATPase inhibitor) reduces transfection (suggesting acidification-dependent endosomal escape).
Toxicity Notes Cell viability (HCT116) decreased in serum-free conditions; in presence of 10% serum, viability ~100% for Tyr2 and Sali; Sali more cytotoxic without serum.
Curation Notes