Back to browse

EXP001688

Paper

Arginine-rich cell-penetrating peptide dramatically enhances AMO-mediated ATM aberrant splicing correction and enables delivery to brain and cerebellum (2011)

Peptide

(RXRRBR)2XB

Sequence: (RXRRBR)(RXRRBR)XB

RNA

AMO (antisense morpholino oligonucleotide)

All experiment fields

Experiment Id EXP001688
Paper Arginine-rich cell-penetrating peptide dramatically enhances AMO-mediated ATM aberrant splicing corr
Peptide (RXRRBR)2XB
Delivery Success Class no
In Vivo Flag no
Uptake Confirmed yes
Label Confidence high
In Vitro Functional Effect yes
Endosomal Escape Evidence
Peptide Concentration 0.5–10 µM CPP-AMO (typical 2.5–5 µM; 4-day treatment for protein assays; 48 h for mRNA assays)
Rna Concentration Same as CPP-AMO (covalent conjugate)
Mixing Ratio Covalent CPP–AMO conjugate (1:1 peptide:AMO)
Formulation Format CPP-conjugated AMO (AVI Biopharma peptide–morpholino conjugate)
Formulation Components (RXRRBR)2XB peptide covalently conjugated to AMO
Size Nm
Zeta Mv
Model Scope in_vitro
Model Type in vitro
Cell Lines Or Primary Cells A-T lymphoblastoid cell line (TATC; homozygous c.7865C>T)
Animal Model
Administration Route
Output Type splice correction + protein restoration
Output Value ≥80% mutant transcript correction at ≥2 µM; ATM protein restored to near WT levels; ATM kinase activity restored (SMC1/KAP1 phosphorylation); radiosensitivity abrogated; ATM detectable up to ~20 days after single 5 µM dose.
Output Units
Output Notes Dose-response 0.5–10 µM; RT-PCR/RT-qPCR, western blot, ELISA, FACS (ATM Ser1981), clonogenic survival assays; prolonged intracellular fluorescence with AMCA tag indicates uptake/retention.
Toxicity Notes No explicit cytotoxicity assay reported for LCLs in vitro; phenotypic rescue observed at 1–5 µM.
Curation Notes