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EXP001995

Paper

Functional peptide nanocarriers for delivery of novel anti-RelA RNA interference agents as a topical treatment of atopic dermatitis (2015)

Peptide

OK-102 (stearoyl-oligopeptide)

Sequence: CHHRRRRHHC

RNA

nkRNA1 (long ssRNA that self-anneals)

All experiment fields

Experiment Id EXP001995
Paper Functional peptide nanocarriers for delivery of novel anti-RelA RNA interference agents as a topical
Peptide OK-102 (stearoyl-oligopeptide)
Delivery Success Class yes
In Vivo Flag yes
Uptake Confirmed no
Label Confidence high
In Vitro Functional Effect
Endosomal Escape Evidence
Peptide Concentration Complex solution applied topically; exact OK-102 amount not explicitly stated (N/P=10).
Rna Concentration 0.375 nmol RNAi agent per administration (nkRNA or PnkRNA) complexed with OK-102; repeated dosing over 2 weeks.
Mixing Ratio OK-102 complexes prepared at N/P (nitrogen:phosphorous) ratio = 10; mixed 30 min at room temperature before use.
Formulation Format CPP peptide/RNA electrostatic complex (cytoplasm-responsive nanocarrier with disulfide crosslinking)
Formulation Components OK-102 (stearoyl-CHHRRRRHHC) complexed with anti-RelA nkRNA1 or PnkRNA1.
Size Nm
Zeta Mv
Model Scope in_vivo
Model Type in vivo
Cell Lines Or Primary Cells
Animal Model NC/Nga mouse model of atopic dermatitis (DNFB-induced ear lesions)
Administration Route Topical transdermal administration to left ear with olive oil cover; 2–3×/week for 2 weeks (days 3,4,7,11,12)
Output Type In vivo therapeutic/phenotypic effect in AD model: ear thickness, clinical score, cytokines (TNF-α, IL-6)
Output Value Suppressed DNFB-induced increases in ear thickness and clinical score; significantly reduced TNF-α and IL-6 vs untreated/scrambled groups.
Output Units
Output Notes Non-invasive topical RNAi therapy for AD-like lesions; no adverse reactions reported.
Toxicity Notes
Curation Notes