Back to browse

EXP001537

Paper

Intracellular delivery of antiviral shRNA using penetratin-based complexes effectively inhibits respiratory syncytial virus replication and host cell apoptosis (2024)

Peptide

Penetratin (PEN)

Sequence: RQIKIWFQNRRMKWKK

RNA

shRNA plasmid (DNA vector)

All experiment fields

Experiment Id EXP001537
Paper Intracellular delivery of antiviral shRNA using penetratin-based complexes effectively inhibits resp
Peptide Penetratin (PEN)
Delivery Success Class no
In Vivo Flag no
Uptake Confirmed yes
Label Confidence high
In Vitro Functional Effect yes
Endosomal Escape Evidence
Peptide Concentration Not reported (prepared at penetratin:plasmid molar ratio 20:1; complexation in HEPES-buffered saline)
Rna Concentration Not reported (50 µL complex added to cells; plasmid dose not explicitly stated in methods excerpt)
Mixing Ratio molar ratio 20:1 (penetratin:plasmid/shRNA vector)
Formulation Format noncovalent CPP/DNA complexes (electrostatic)
Formulation Components Penetratin + pGFP-V-RS (RSV F-shRNA + GFP) plasmid
Size Nm 164.00
Zeta Mv 8.70
Model Scope in_vitro
Model Type in vitro
Cell Lines Or Primary Cells HEp-2 (human laryngeal epidermoid carcinoma) infected with RSV A2 (MOI 0.1)
Animal Model
Administration Route
Output Type Antiviral activity (RSV F gene copy number by qPCR; viral titer by plaque assay) + transfection efficiency (GFP, flow cytometry)
Output Value Transfection efficiency ~93% (48 h); RSV F gene copies reduced from 8.8×10^5 (untreated) to 5.45×10^3; viral titer reduced from 1.8×10^6 PFU/mL to 8.3×10^4 PFU/mL (24–48 h windows as reported)
Output Units
Output Notes Complexation optimal at 20:1 by gel shift; penetratin-shRNA reduced syncytia/CPE, viral RNA (F gene), and progeny virus production in HEp-2 supernatants.
Toxicity Notes No significant cytotoxicity vs untreated in HEp-2 (MTT) for penetratin-shRNA (20:1 and 25:1) up to 72 h.
Curation Notes