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EXP001539

Paper

Intracellular delivery of antiviral shRNA using penetratin-based complexes effectively inhibits respiratory syncytial virus replication and host cell apoptosis (2024)

Peptide

Penetratin (PEN)

Sequence: RQIKIWFQNRRMKWKK

RNA

shRNA plasmid (DNA vector)

All experiment fields

Experiment Id EXP001539
Paper Intracellular delivery of antiviral shRNA using penetratin-based complexes effectively inhibits resp
Peptide Penetratin (PEN)
Delivery Success Class no
In Vivo Flag no
Uptake Confirmed yes
Label Confidence high
In Vitro Functional Effect yes
Endosomal Escape Evidence
Peptide Concentration Not reported (penetratin:plasmid molar ratio 20:1; 50 µL/well used in apoptosis assays)
Rna Concentration Not reported
Mixing Ratio molar ratio 20:1 (penetratin:plasmid)
Formulation Format noncovalent CPP/DNA complexes (electrostatic)
Formulation Components Penetratin + RSV F-shRNA plasmid (pGFP-V-RS)
Size Nm 164.00
Zeta Mv 8.70
Model Scope in_vitro
Model Type in vitro
Cell Lines Or Primary Cells A549 (human lung epithelial carcinoma) infected with RSV A2 (MOI 0.1)
Animal Model
Administration Route
Output Type Apoptosis-related readouts (DNA fragmentation gel; PI staining; caspase 3/7 activity)
Output Value Reduced DNA fragmentation and apoptotic morphology; caspase 3/7 activity significantly reduced vs untreated RSV-infected cells
Output Units
Output Notes In RSV-infected A549 cells, penetratin-F-shRNA mitigated apoptosis markers and reduced caspase 3/7 activity; GFP microscopy confirmed successful transfection.
Toxicity Notes No penetratin/shRNA-specific toxicity reported in cell viability assays (HEp-2); A549 data focus on apoptosis markers during infection.
Curation Notes